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The Essentials On Mitochondrial Vitamins

By Tracie Knight


The global health research shows that most people do not have sufficient intake and access to Vitamin D. Deficiency in affected individuals are characterized with rickets formation and muscle strains. The mitochondrial vitamins C and D are fundamental remedy for such patients. The vitamin D is gotten mostly from sun energy. It aids in bone formation thus caretakers of young children are always advised to expose the infant to sun energy at given moments. Foods such as fish liver oil and fish are also sources.

The bodily movement of the human being is assisted by the functioning of the mitochondria. This has been implied to be some sort of battery cells in human body. This cell organelle utilizes glucose and oxygen that are mechanically broken down to provide energy for muscle movement. Hence, deficiencies in vitamin D will lead to none functioning of cell organelle thus arise of diseases such as rickets.

The Adenosine Triphosphate usually abbreviated as ATP is the form of energy that is adamant on the movement of human muscles. The cells in the muscles use the hormone phosphocreatine to produce the ATP. The mitochondria aids the regeneration of this phosphocreatine once the muscles contract. The low levels of Vitamin D affect the functions of this cell organelle.

Recent researches are on the look for health issues that are emerging based on its role in human body. They have far considered a test on patients, where a dosage was given orally for a period of ten to twelve weeks. It was noted that the supplements in the oral dosage given, led to a decrease in the functioning of the mitochondria and time recovery on the phosphocreatine.

A dual relation of oxidized vitamin C in the organelle is still on the research for clear relationship. This nutrient is associated with prevention of diseases such as scurvy, gum diseases. The likelihood of tracing its existence in the mitochondria is on the glucose GLUT10 that transports it and function to protect the cell from oxidative stress.

This finding increased understanding on causes of hereditary connective tissue disorder of arterial tortousity syndrome. It was also deduced that oxidative stress occurs due to cellular imbalance of molecules that are chemically reactive that contain reactive oxygen species and the ability of unit of life to detoxicate the unit of life and help in the repair of damaged tissues.

Predictions of mitochondria being the production site of reactive oxygen species is by most scientists. They believe that it also helps in recycling of dehydroascorbic acid from its activity. Correlation on molecular means, that are based on dehydroascorbic acid is still to be unraveled. However, it has been noted that cases of arterial tortousity syndrome is due to abnormal functioning of GLUT10. This can also be applied in understanding the relation of type 2 diabetes and GLUT10 as a corroboration of ROS and DHA.

Human body require nutrients to function properly, the need to take a diet meal will help in body fight pathogens on its own natural mechanism. Improving your immunity is also of much use since administration of drugs is not that efficient. Then if this is so then why waiting till you contract the disease yet you have the preventive measures at hand. These measures range from massive intake of fruits and vegetables on a diet basis.




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